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Population and the Environment

Ozone Depletion and Human Health

  • 3 things to know
  • Component 2

Overview

Recall questions on why thinner stratospheric ozone admits more ultraviolet radiation and on the path from DNA damage to skin cancer risk.

What you need to know

Answer each one before you reveal it.

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Key term

Why does reduced stratospheric ozone lead to greater ultraviolet radiation reaching the Earth's surface?

Key term

What is the link between DNA damage in skin cells and skin cancer risk?

Key term

What type of damage does UV radiation cause to lens proteins in the eye?

Questions from this subtopic

Each one with the model answer underneath.

  1. 1

    Why does reduced stratospheric ozone lead to greater ultraviolet radiation reaching the Earth's surface?

    Model answer

    Stratospheric ozone normally absorbs UV-B radiation; when ozone is depleted, less UV-B is filtered out, allowing more to penetrate to the surface.

  2. 2

    How does increased UV-B radiation penetration cause cellular harm in the skin?

    Model answer

    UV-B radiation damages DNA in skin cells, causing mutations that can disrupt normal cell function and replication.

  3. 3

    What is the link between DNA damage in skin cells and skin cancer risk?

    Model answer

    Accumulated DNA mutations from UV exposure can lead to uncontrolled cell division, elevating the risk of developing skin cancer.

  4. 4

    How does ozone depletion ultimately elevate skin cancer risk in human populations?

    Model answer

    Reduced ozone allows more UV-B to reach the surface, causing DNA damage in skin cells, which increases the likelihood of skin cancer development.

  5. 5

    Why does increased UV exposure from ozone depletion affect the eye?

    Model answer

    Higher levels of ultraviolet radiation reach the eye, where prolonged exposure can damage sensitive ocular tissues.

  6. 6

    What type of damage does UV radiation cause to lens proteins in the eye?

    Model answer

    UV radiation causes oxidative damage to lens proteins, altering their structure and function.

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