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Autistic Spectrum Behaviours

Autistic Spectrum Behaviours: characteristics, biological and individual differences explanations

  • 3 things to know
  • A2 Unit 3 Section A

Overview

The two core domains and sensory differences in autism, the amygdala, chloride and genetic explanations, gender differences in diagnosis, the Sally-Anne task and weak central coherence theory.

What you need to know

Answer each one before you reveal it.

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Numbers

What evidence points to a genetic predisposition to autism?

Key term

What gender differences appear in autism diagnosis, and why?

Key term

What are the two core domains used to characterise autistic spectrum behaviours?

Questions from this subtopic

Each one with the model answer underneath.

  1. 1

    What are the two core domains used to characterise autistic spectrum behaviours?

    Model answer

    Differences in social communication and social interaction, and restricted or repetitive patterns of behaviour, interests and activities; the profile varies so widely between individuals that it is described as a spectrum.

  2. 2

    What sensory differences are commonly reported by autistic people?

    Model answer

    Both hyper-reactivity and hypo-reactivity to sensory input: adverse responses to particular sounds or textures and excessive smelling or touching of objects on the one hand, and apparent indifference to pain or temperature on the other, alongside fascination with lights or movement.

  3. 3

    What does the amygdala theory of autism propose?

    Model answer

    Baron-Cohen et al. (2000) proposed that the amygdala, part of the social brain, develops and functions differently in autistic people, contributing to differences in processing faces, eye gaze and emotional expression.

  4. 4

    What does the chloride ions at birth explanation of autism propose?

    Model answer

    Tyzio et al. (2014) found that a surge of oxytocin during birth normally lowers intracellular chloride so that GABA switches from excitatory to inhibitory; in rodent models of autism this switch failed, leaving GABA excitatory.

  5. 5

    Which drug was used to test the chloride hypothesis, and what did it do?

    Model answer

    Bumetanide, which blocks chloride entry into the neurone, restored the GABA switch and reduced autistic-like behaviours in the rodent offspring.

  6. 6

    What evidence points to a genetic predisposition to autism?

    Model answer

    Twin studies from Folstein and Rutter (1977) onwards find much higher concordance in monozygotic than dizygotic pairs, and siblings of autistic children are more likely to be autistic; the pattern is polygenic rather than the work of any single gene.

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