OCR A-Level Psychology H567
Research Methods and Techniques
Planning and Conducting Research
Data Recording, Analysis and Presentation
Report Writing and Scientific Communication
Practical Activities and the Nature of Science
Mathematical Requirements for Research Methods
Studying and Evaluating the Core Studies: The Common Framework
Core Studies: Social Area
Core Studies: Cognitive Area
Core Studies: Developmental Area
Core Studies: Biological Area
Core Studies: Individual Differences Area
Areas and Perspectives in Psychology
Debates in Psychology
Practical Applications of Psychology
Issues in Mental Health
Child PsychologyApplied option
Criminal PsychologyApplied option
Environmental PsychologyApplied option
Sport and Exercise PsychologyApplied option
Methodological Issues and Debates Running Through Applied Psychology
Issues in Mental Health
The biochemical explanation of mental illness
- 3 things to know
- Component 03
Overview
Recall questions on the medical model's central assumption, the biochemical explanation, the original and revised dopamine hypotheses and their evidence, the monoamine hypothesis, cortisol in depression, the delay in antidepressant action, and the treatment aetiology fallacy.
What you need to know
Answer each one before you reveal it.
Which hormone is found at raised levels in many depressed patients?
Why is the time course of antidepressant action a problem for the biochemical explanation?
Questions from this subtopic
Each one with the model answer underneath.
- 1
State the central assumption of the medical model.
Model answer
It assumes that mental disorders are illnesses with physical causes in the body, particularly the brain, and should be diagnosed and treated in the same way as any other physical illness.
- 2
What does the biochemical explanation propose?
Model answer
It proposes that mental disorders result from imbalances in neurotransmitters or hormones which disturb the way messages pass between neurons.
- 3
What does the original dopamine hypothesis claim?
Model answer
It claims that excessive dopamine activity, or oversensitive dopamine receptors, in subcortical areas such as the limbic system produces the positive symptoms of schizophrenia.
- 4
How does the revised dopamine hypothesis account for negative symptoms?
Model answer
It proposes that dopamine activity is abnormally low in the prefrontal cortex at the same time as it is abnormally high in subcortical areas, and this underactivity produces the negative symptoms.
- 5
Give two pieces of evidence supporting the dopamine hypothesis.
Model answer
Amphetamines raise dopamine activity and can produce psychotic symptoms in healthy people, and the drugs that reduce positive symptoms work by blocking dopamine receptors.
- 6
What does the monoamine hypothesis say about depression?
Model answer
It says depression is caused by low levels or low activity of the monoamine neurotransmitters, particularly serotonin and noradrenaline.
Other ways to work this
The same content, four other ways.
What is inside Levo
Six ways to work the same material.
Every question in Issues in Mental Health, on a schedule.
Attempt a question from memory, write the correct version, then attempt it again. Levo records the jump between your first attempt and your last, and brings each question back on the day you are due to forget it.
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